BHA natural Salicylic Acid

Natural ß-hydroxy acid, exfoliating, antimicrobial and anti-acne active, contributes significantly to cell regeneration and skin renewal, acts against microbes involved in acne formation. It is a natural and gentler alternative to synthetic salicylic acid, a standardised extract from black willow bark with proven efficacy, ideal for clear, smooth and younger-looking skin.

Provides the benefits of salicylic acid such as exfoliation and anti-microbial action, without any of the typically associated irritation from the synthetic version.

BENEFITS

• standardized for 10% natural salicylates
• alternative to synthetic salicylic acid
• ideal for reducing inflammation and associated redness
• acts as an efficacious antimicrobial agent
• exfoliating agent, keratolytic
• anti-dandruff due to its keratolytic and purifying activity
• purifies the skin in depth
• potent antioxidant, anti-inflammatory, anti-aging
• sebum control activity, clear pores, helps fight blackheads and tightens pores
• enhances cell renewal
• boost collagen synthesis
• improves skin density

USE RECOMMENDATIONS 

• cosmetic ingredient
• can be used in any cosmetic formulation to enhance cell turnover, as a natural anti-microbial agent, or as a replacement for synthetic salicylic acid
• ideal for problem skin and scalp applications
• suggested use levels 2.0 - 10.0% and up to 20% for peeling solutions
• optimal pH of the finished product 4.0 - 7.0
• add to water phase, stable up to 80°C

SAFETY DATA

• cosmetic use only
• for professional use refer to safety datasheet 

EFFICACY TESTS

• Cellular Renewal Assay
• Provided better cellular renewal than the 1% synthetic salicylic acid; showed a 26% increase, while the synthetic showed a 22% increase.
• High Resolution Skin-Imaging Assay
• Improved skin density by 36.72% after 24 hours and after 4 weeks improved density by 32.41%.
• Anti-microbial Assay
• The results of the antibacterial assay indicated significant activity against all of the gram-positive bacteria tested with minimum inhibitory concentrations ranging from 0.1 to 5.0 mg/ ml.